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This broad tissue distribution explains why GLP-1 RAs exert effects on multiple organ systems simultaneously, a property that makes them particularly suited to address the complex pathophysiology of NASH, which involves dysregulation of glucose and lipid metabolism, insulin resistance, inflammation, and fibrosis

Preclinical research suggests it may support tissue repair, gut healing, and inflammation reduction by increasing VEGF (vascular endothelial growth factor) expression and promoting new blood vessel growth at injury sites

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