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doi: 17
Regulates actin polymerization, promotes angiogenesis, reduces inflammation at the injury site
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Glutathione might inhibit the effects of cerebral infarction and enhance antiapoptotic signaling after ischemic stroke, suggesting that GSH may be a potent therapeutic antioxidant that can attenuate severe pathologies after ischemic stroke, and stimulating GSH synthesis through administration of GSH precursors and micronutrients like selenium can optimize GSH and GSH peroxidase for optimal antioxidant defense in cerebral ischemia (155, 156)
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Tolerant lines maintained better growth and antioxidant defense, whereas susceptible cultivars accumulated higher levels of oxidative damage (hydrogen peroxideH 2 O 2 and malondialdehydeMDA)
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