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For treatment of primary systemic carnitine deficiency, a genetic impairment of normal biosynthesis or utilization of levocarnitine from dietary sources, or for the treatment of secondary carnitine deficiency resulting from an inborn error of metabolism such as glutaric aciduria II, methyl malonic aciduria, propionic acidemia, and medium chain fatty acylCoA dehydrogenase deficiency

This inactive roGFP2 construct was expressed in HeLa cells and subjected to the same 48-hour timelapse imaging ( Figure 3b )

Dose discipline: Adhere strictly to conservative dosing

Custom values become necessary when working with non-standard vial sizes, specific concentration targets, or when you need precise control over the final solution strength

The physiological role of mitochondrial calcium revealed by mice lacking the mitochondrial calcium uniporter

Hays RD, Sherbourne CD, Mazel RM