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In turn, those SCFAs signal cells in the gut to release GLP-1 into the bloodstream
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Furthermore, gene-editing therapies targeting Angiopoietin-like 3 (ANGPTL3) 399,400 and LDLR 401,402 have entered the preclinical stage, highlighting the broader potential of gene-editing-based lipid-lowering strategies, and long-acting therapies that provide stable and durable lipid-lowering effects could further reduce the risk of cardiovascular events
In addition to its intrinsic role in catalysing glycolysis, PFKFB3 was recently reported to be involved in NLRP3 inflammasome-mediated inflammation, oxidative stress, and pyroptosis during sepsis [52]
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