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(54) found that administering GIPR antagonist to healthy humans reduced postprandial insulin secretion and increased glucose excursions more than administering GLP-1R antagonist (GLP-1RA), indicating that endogenous GIP may play a more significant role in stimulating insulin secretion and reducing postprandial plasma glucose excursions compared to endogenous GLP-1
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10.1093/neuonc/noy131 124 OussalahA.RischerS.BensenaneM.ConroyG.Filhine-TresarrieuP.DebardR.et al (2018)
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