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Ros generation and autophagosome accumulation contribute to the dmamcl-induced inhibition of glioma cell proliferation by regulating the ros/mapk signaling pathway and suppressing the akt/mtor signaling pathway

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In the cblC, cblD, cblF, cblG, cblJ, and cblX group of disorders, the synthesis of both adenosylcobalamin and methylcobalamin are affected, leading to combined methylmalonic aciduria and homocystinuria

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