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doi: 10.1111/j.1752-8062.2008.00070.x 209 RuffellBChang-StrachanDChanVRosenbuschAHoCMPryerNet al
Chong, P
Programmed necrosis in cardiomyocytes: mitochondria, death receptors and beyond
Figure 2C highlights the immunological arm of this axis, showing how SCFAs and related metabolites polarize macrophages toward an M2-like phenotype, promote Treg differentiation and increase IL-10/TGF- production, thereby dampening systemic and islet inflammation and indirectly preserving -cell function
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