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& Sutton, G
Glucagon receptor agonism adds a component to the profile of the molecule increased energy expenditure in the metabolic literature, glucagon is studied as a signal intensifying thermogenesis and hepatic lipolysis
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However, it is not clear what contributed specifically to this effect on inflammation, though increasing evidence suggests that IGF-1 signaling is critical for maintaining immune cell homeostasis 44 , but the prospects of sex differences in the requirement for IGF-1 signaling in these cells is not yet known and will require further study
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