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GLP-1 receptor agonists (GLP-1RAs), primarily developed for their metabolic effects 6,7,8 , demonstrated neuroprotective activity in preclinical models over two decades ago 9 with subsequent studies revealing anti-inflammatory effects in both central and peripheral tissues 10

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This regulated necrotic cell death is activated in response to Fas/FasL signaling, CD59 ligation combined with pore formation, mediated by ROS, ceramide, and advanced glycation end products (AGEs) [28]

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