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These findings compel further research to understand how mitochondrial metabolism and the related epigenetic programming of brain plasticity can serve as new targets to develop mechanism-based treatment models for psychiatric and neurodegenerative diseases
Decreases inflammation: Can suppress chronic inflammatory cues without suppressing immune function strongly
Loads of animal studies have given credence to so-called anti-aging interventions interventions that slow aging
Clinical Drug Investigation
went all out the window
Methylmercury chloride exposure affects oocyte maturation through AMPK/mTOR-Mediated mitochondrial autophagy
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