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The underlying mechanisms may be associated with elevated myocardial MOTS-c levels, which in turn activates AMPK, increases Keap1 levels, and enhances Nrf2 expression
113, E4966E4975
A reduction in CYPOR protein levels prevented NFT-dependent accumulation of Nrf2 protein and partially attenuated the NFT-dependent increase in glutathione levels
By tailoring NPs to specific therapeutic objectives, such as mitigating oxidative stress or reducing inflammation, researchers are developing more effective and targeted AD treatments [205]
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