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Therapeutic targets for endothelial dysfunction in vascular diseases

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Acknowledgements This work was supported by the Japan Society for the Promotion of Science (JSPS) KAKENHI (Grant Numbers JP20H05626 (H.S.), JP25H00970 (H.S.), JP23H02076 (F.N.), JP24K01641 (K.O.), JP23KJ1331 (E.M.)), the Japan Science and Technology Agency (JST) CREST (Grant Number JPMJCR23B3 (H.S.)), JST FOREST program (Grant Number JPMJFR2002 (K.O.)), iPS Cell Research Fund from Centre for iPS Cell Research and Application (Kyoto University)

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GSH moderates the ISR gene expression during cystine starvation Activation of the ISR elevates ATF4 to increase expression of genes which contain the amino acid response elements (AARE) 16