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In 2025, we explored how GLP-1s were beginning to reshape consumer behavior, identifying early evidence of reduced food consumption and shifting demand patterns

however, organ damage during MTX treatment reduces its therapeutic effectiveness (Reference Shinde, Venkatesh and Kumar32)

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The intermediates from de novo lipogenesis in the liver will disrupt mitochondrial function and produce ROS, and then cause endoplasmic reticulum (ER) stress, ultimately leading to inflammation, and the broken cells releasing cytokines will cause more cell death ( The excess free fatty acids (FFAs) and de novo lipogenesis would break the homeostasis of lipid metabolism

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In such patientsespecially those with heart or kidney diseasethese medications improved blood glucose control, reduced the risk of cardiac and renal complications, promoted weight loss, and lowered the risk of premature death