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Thus, the extent to which endometriosis causes oocyte aneuploidy remains to be answered definitively, and if it does, the extent to which the aneuploidy is caused by ROS in endometriosis will need to be addressed

Collectively, these data indicated that the more pronounced weight loss phenotype of PAR2-G37I versus PAR2-R38E mice is not explained by differences in ATM or adipose tissue endothelial cell reprogramming

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Patient follow-up completes the support need