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Studies using cardiac-specific Atg5 deletion models have been pivotal in establishing this concept (Fig
Therefore, our objective was to explore the interplay between two key players in BD disease pathophysiology: mitochondrial dysfunction and inflammation
With regard to catalytic mechanism, all the seleno-GPxs share the common pathway which starts with the binding of hydroperoxides and GSH at the catalytic and thiol binding motifs, respectively [8, 11,12,13,14,15,16,17,18]
Archived from the original on 9 January 2022
22 Taken together, these mixed results tell us that we need to be careful about picking the right patients, develop good biomarkers, and pay attention to drug specific properties in future studies
