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Overall, we conclude that PC patients with low expression of VRK2 are sensitive to GSH-targeting therapy, while PC patients with high expression of VRK2 are re-sensitive to GSH-targeting therapy following VRK2 inhibition, indicating genetic aberrance of VRK2 expression stratifies the response of PC to metabolism-targeted therapies

R.et al (2017)

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The peptide stimulates VEGF (vascular endothelial growth factor) and HGF (hepatocyte growth factor) production, both of which support blood flow to hair follicles

D., Gaillard E

These limitations have direct clinical implications