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Description

TBI brain damage involves mechanisms of acute cerebrovascular disease and ferroptosis-related chronic NDDs

Tryptophan 33 in GLP-1R is required for PF-06882961 signaling Prior to the selection of animal models for in vivo pharmacology studies, we characterized the potential for species differences in GLP-1R activation with our small-molecule agonists

Hsieh MJ, et al

All mice were housed and treated in compliance with the European Council directives (No.86/609/EEC) and with NIH Guide for the care and use of laboratory animals (eight edition)