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Metabolites such as HMGB1 and glutamate released by dead cells can reshape the immune microenvironment through a dual mechanism (153): HMGB1 binds to TLR4 on the surface of DCs, activates the NF-B pathway to promote DC maturation and MHC molecule expression, and then activates tumor antigen-specific CD8+ T cells
doi: 10.1111/acer.15032
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After continuous research by scientists, various new pathways of ferroptosis independent of GPX4 have been discovered
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