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The results demonstrated that the liver of diabetic mice had a higher phosphorylation level of NF-B, which was reduced by NaB and CB (Fig
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Mass spectrometry and amino acid analysis verify the Trp-Ala-Gly-Gly-Asp-Ala-Ser-Gly-Glu sequence and 99%+ purity
Cholesterol-stimulated INS-1 cells were cultured with GLP-1 with the pre-treatment of AMPK activator AICAR or AMPK inhibitor Compound C to examine whether AMPK activation could repress PARP1 activity and consequent PARylation
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