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Current AD research focuses on understanding pathology by targeting several mechanisms, such as abnormal tau protein metabolism, -amyloid, inflammatory response, and cholinergic and free radical damage, aiming to develop successful treatments that are capable of stopping or modifying the course of AD (Tamagno et al., 2021)

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Tissue damage in the amyloidoses: Transthyretin monomers and nonnative oligomers are the major cytotoxic species in tissue culture

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