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Moreover, R196 ECL1 Y diminished retatrutide potency by 107.7-fold, whereas P195 2.72b K markedly decreased GIPR-mediated cAMP accumulation, supporting a key role of GIPR ECL1 in retatrutide agonism (Fig
7b-c, 1.08 0.19 vs
19 This discrepancy highlights the gap between efficacy observed in controlled trial settings and effectiveness in routine clinical practice
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