doi: 10.1021/jacs.0c06379 121 WangSLiaoHLiFLingD
Interestingly, the GltT-defective mutant also exhibited less adhesive and invasive properties to human bronchial epithelial cells, and transcriptome analysis suggested that genetic inactivation of gltT led to remodeling of the outer membrane and surface structures (Li et al., 2009)
In vitro functional analyses confirmed their mature hepatocyte characteristics: they showed high transcription and protein expression of ALB, A1AT, CYP3A4, and CYP2C9, comparable to primary human hepatocytes (PHs)
Effects of liraglutide on beta-cell-specific glucokinase-deficient neonatal mice
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