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The result is a 16-amino acid peptide that stimulates lipolysis through beta-3 adrenergic receptors on fat cells, without triggering IGF-1 elevation, insulin resistance, or the growth-promoting effects that make full HGH problematic for long-term metabolic use
We previously showed that FXR is expressed in enteroendocrine L-cells where it regulates GLP-1 production in response to glucose 12
10.1210/en.2011-1178 44 DrabS
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