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In vitro , low mM concentrations (e.g., 8.5 mM) reduce TEER and increase permeability across Caco-2 monolayers without compromising epithelial integrity, suggestive of a paracellular mechanism (Lindmark et al., 1995)
Furthermore, the protection of tea polyphenols is more potent than that of sulfhydryl group-containing compounds, including GSH [26]
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An alternative is to develop substances that are safer for the epithelial barrier and are substitutes for commonly used, dangerous compounds [1]
Preclinical studies have explored its involvement in repair processes, vascular adaptation, and inflammatory regulation, positioning it as a molecule of interest in musculoskeletal and gastrointestinal research contexts (1)
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