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Viewed from this perspective, mTORC1 is a pro-oncogenic factor as it represses autophagy by either directly inhibiting multiple stages of autophagy or indirectly regulating lysosomal biogenesis (Rabanal-Ruiz et al., 2017) (Figure 2)
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Maximal reduction of liver fat appears to require both inhibition of hepatic de novo lipogenesis and suppression of adipose tissue lipolysis (51, 52)
Bkdal TA, Donsmark M, Hartoft-Nielsen ML, Sndergaard FL, Connor A
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Metabolism 2011;60(4):480-485
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