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Targeting ACSL4-dependent lipid remodeling or enhancing FSP1-mediated antioxidant defense provides additional strategies to modulate ferroptosis in disease contexts

Singh et al., 2021)

Researchers investigate this combination to better understand interactions between melanocortin receptor biology, GABAergic signaling, intracellular communication, neuroimmune pathways, and peptide pharmacology within controlled laboratory environments

2006, Philadelphia: Lippincott, Williams and Wilkins, 537-544

HIV-1 Tat expression and sulphamethoxazole hydroxylamine mediated oxidative stress alter the disulfide proteome in Jurkat T cells