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Both peptides thus address complementary aspects of vessel formation: TB-500 via the intracellular actin dynamics of the migrating cells, BPC-157 via the receptor-mediated VEGFR2-NO axis

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suggest that the peptides upregulation of cellular movement may mirror the early steps in angiogenesis, where living cells extend and organize into primitive vessel structures to facilitate tissue repair

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the mechanistic rationale (BPC-157 favouring fibroblast and NO-mediated repair, T4 favouring actin dynamics, cell migration and angiogenesis) is entirely extrapolated from separate animal experiments