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Because its exact etiology remains largely unclear, and the primary therapy is limited to symptomatic treatment, the development of new therapeutic agent for UC is highly desired

GLP-1(7-37) represents 98%)

BPC-157 significantly promotes angiogenesis by enhancing vascular endothelial growth factor receptor-2 (VEGFR2) activity and nitric oxide signaling through activation of the Akt-endothelial nitric oxide synthase (eNOS) pathway

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[Google Scholar] Distinct metaplastic and inflammatory phenotypes in autoimmune and adenocarcinomaassociated chronic atrophic gastritis