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The PINK1/Parkin pathway is triggered in response to mitochondrial damage, resulting in the recruitment of Parkin to impaired mitochondria and subsequent mitophagy

We found that deletion of IGF1R in Nestin + MSCs affected the differentiation, but not migration of MSCs during bone remodeling

Interestingly, a comparison of acute metabolic responses with the chronic profiles at the end of the treatment period strongly suggests that while the different GLP1-based agonists may share primary mechanisms of action, the durability and efficiency of their long-term metabolic responses differ, with clear superiority for the GLP1/E compound

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