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Unlike native IGF-1, the LR3 variant does not bind strongly to IGF Binding Proteins (IGFBPs)

Such binding may displace other regulatory proteins, making DNA more accessible for transcriptional machinery, potentially inducing neuronal cell differentiation in retinal and periodontal ligament stem cells (hPDLSCs)

Given its prominent proangiogenic effects, deciphering the mechanistic basis of its vascular regenerative actions is critically important