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Despite the limited data from human trials, current studies have focused on specific molecular mechanisms and used representative animal models of PD
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Part D, conversely, is for drugs the patient administers(filled at pharmacy)
These genetic factors also interact with GIPR variants, creating a unique peptide-pathway signature for each individual
This was further supported by experiments in the SAMP8 mouse model of AD, in which liraglutide delayed or halted the decline in memory function along with hippocampal neuronal loss [47]
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