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It actively binds to and neutralizes highly reactive and damaging oxidant species, including nitrogen dioxide and hypohalous acids, providing an immediate and critical buffer against widespread tissue damage

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The relationship between dose, route of administration, and clinical outcomes requires systematic investigation to establish therapeutic ranges and maximize efficacy while minimizing potential risks

4-HNE represents a highly reactive lipid peroxidation byproduct that induces cellular damage by interacting with proteins, DNA, and other cellular components through iron- and oxygen-mediated chain-breaking reactions ( The transient receptor potential channel TRPM4 has been shown to promote ox-LDL-induced dysfunction in HCAECs through ferroptosis mechanisms (33)

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