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Based on prior reports linking mitochondrial protein stability to a short fragment within the cyclic structural domain of SLC25A39 (amino acids 7286) that mediates glutathione (GSH) response and recruits SLC25A39 to AFG3L2 for proteolysis, we generated a deletion mutant lacking this fragment (aa72-86), confirmed by Sanger sequencing (Fig

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Research indicates that 1% is the clinical "sweet spot" for mature skin

In the normal pancreas specimen, no -SMA-positive PSCs (activated myofibroblast-like cells) were seen, however, GFAP-positive quiescent PSCs showed low expression of the GLP-1R (Figures 1 eg)

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