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Another mechanism of GC-mediated inhibition of osteoblast differentiation is their direct interference with the Wnt/-catenin signaling [27, 28]
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In the liver, it decreases glucose production, while in the pancreas it promotes insulin release and beta-cell proliferation
Any disruption to this pathway changes the risk profile of exogenous copper administration
GLP-1 agonists may be useful in critically ill patients in this regard
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