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Abstract Background Oxidative stress (OS) is an imbalance between oxidant and antioxidant species and, together with other numerous pathological mechanisms, leads to the degeneration and death of motor neurons (MNs) in amyotrophic lateral sclerosis (ALS)

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These systems improve drug delivery efficiency and specificity at the targeted region by using receptor endocytosis, enzyme-responsive drug release, and pH-sensitive drug triggering

Presented for research literature review only Zou W et al

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