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Published research has examined its role as an exercise-induced regulator of metabolic homeostasis and physical capacity, with downstream effects on the AMPK / folate-methionine cycle

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It was reported that phosphorylation of DET1 Ser458 by MAPK inhibits CRL4 COP1/DET1 by an as yet unknown mechanism 57

This review revises and expands the metabolic-rescue concept for cell therapy in ischemic stroke

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