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In mice GLP-1R is expressed in the atrium of the heart and GLP-1R agonist promotes the secretion of atrial natriuretic peptide [17], which consequently causes diuresis and natriuresis
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GLP-1R siRNA (50 nM) or GSK3 siRNA (50 nM) was transfected using Lipofectamine RNAiMAX reagent (Invitrogen), according to the manufacturers instructions
The research on this intersection is early
The study analyses were conducted using SAS version 9.4 (Cary, NC)
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