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Because GHK-Cu is studied as a naturally declining protective factor, with research examining its roles in maintaining extracellular matrix integrity, protecting against oxidative stress, and supporting tissue repair capacity, it is a central subject in experiments related to age-related tissue changeswithout implying outcomes outside of a research setting.

We revealed that overexpression of GSTM3 could inhibit the ROS level and reduce the level of glycolysis in PC cells, and the opposite outcome was presented in the knockdown group