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(2016) "Neuroprotective effects of GHK-Cu on oxidative stress-induced neuronal cell death." Published in Neuroscience Letters
The investigation further examines how GHK-Cu interacts with markers of oxidative stress, such as malondialdehyde (MDA) and glutathione (GSH)
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GSH is the most important antioxidant and free radical scavenger that is found to be decreased in the brains of people with a wide range of mental and neurological illnesses from schizophrenia[iv] to dementia[v] [vi]
O., Gerke-Duncan, M
Cardiovascular Protection Myocardial infarction Protected against isoprenaline-induced MI in rats, with reduced cardiac damage markers Pulmonary hypertension Both prevented and reversed monocrotaline-induced pulmonary hypertension in rats Arrhythmias Antiarrhythmic effects, including counteracting digitalis-induced ventricular tachycardia Thrombosis Prevention and reversal demonstrated in animal models (PMID: 36359218) Liver Protection Hepatoprotective effects Protected against liver damage from restraint stress, bile duct ligation, hepatic artery ligation, and carbon tetrachloride (CCl4) toxicity (PMID: 7901724) Radiation-induced liver injury Reduced apoptosis and lipid accumulation via KLF4 upregulation (PMID: 36228773) Alcohol-related damage Attenuated liver lesions in chronic alcohol models For practical information on how researchers have applied these findings, see our BPC-157 Dosing Guide and BPC-157 Results Timeline
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