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The association between GLP-1s and wet AMD is less clear, with different studies showing conflicting results, says Rachitskaya

Lichen sclerosus

These SCFAsparticularly butyrate, propionate, and acetatebind to receptors on L-cells, triggering GLP-1 release

MBOAT1/2 inhibit ferroptosis by remodeling the cellular phospholipid profile, and strikingly, their ferroptosis surveillance function is independent of GPX4 and FSP1

This creates optimal balance between synthesis and degradation, enabling damaged matrix to remodel into healthier architecture

doi: 10.1038/aps.2011.168, 59 HanssenR.RigouxL.KuzmanovicB.IglesiasS.KretschmerA