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In addition to being a therapeutic target, oxidative stress has been shown to decrease the GSH:GSSG ratio which has proven useful as a clinically accessible, pharmacodynamic biomarker of immune redox tone for conditions such as malignancy (Zitka et al., 2012)

This increase was due to an enhanced activity of GSH1, the key enzyme in glutathione synthesis, observed both in CO23RFLC and in CO23RFLC treated with FLC

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Zinc selectively blocks neurosteroid-sensitive extrasynaptic GABAA receptors in the hippocampus

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The glutathione synthesis pathway is destroyed, and the oxidative damage in the cells increases, which leads to apoptosis and inhibits tumor growth