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It is known to enhance growth factor signaling (e.g., increasing receptors for VEGF and PDGF) and upregulate nitric oxide pathways, leading to improved microcirculation in injured tissue [5]
In addition to this, GLP-1RAs may directly improve hepatic steatosis via the upregulation of fatty acid metabolism and insulin signalling pathways [12]
Interestingly, the strong dependence of mitochondrial -oxidation on autophagic and fatty acid (FA) catabolic processes makes some tumors more resistant to nutrient deprivation and environmental stressors 13
Despite the differences in the findings of these studies ( Table 2 ), the results do suggest that long-term (6 months) and adequate-dose vitamin D supplementation may reduce SF in patients with DRE, but this requires confirmation in larger, prospective trials
1 2 3 4 JAMA Oncology
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