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Recent methodological advancesincluding organotypic slice cultures, three-dimensional brain organoids and in vivo imaging approachesoffer scalable and physiologically relevant platforms that more faithfully capture astrocyte heterogeneity and functional consequences
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They enabled enrollment of patients most likely to harbor the target pathology
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The N-terminal domain of BNIP3, a mitochondrial protein, is up-regulated in response to hypoxia and anchored to the OMM through its C-terminal transmembrane (TM) domain, exposing the N-terminal domain to the cytoplasm [11]
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