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Cystine/glutamate antiporter and aripiprazole compensate NMDA antagonist-induced dysfunction of thalamocortical L-glutamatergic transmission

30 June 2014

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The rationale for combining them is based on the hypothesis that parallel modulation of nitric oxide signaling, cytoskeletal dynamics, extracellular matrix remodeling, and oxidative stress responses may generate additive or synergistic effects in controlled experimental models

Figure 1 2.3 Oxidative stress-mediated reproductive dysfunction Excessive accumulation of ROS disrupts reproductive function by impairing gamete integrity, altering hormonal signaling, and promoting chronic inflammation (Figure 1, highlighting the systemic nature of ROS-induced reproductive failure