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It is well acknowledged, VEGF expression is reduced in diabetic CC, affecting VEGF-mediated signaling, decreasing endothelial NOS activation, and reducing endothelial cell viability [84]
Unlike most abundantly synthesized molecules, such as ATP or DNA, the production of GSH is controlled at the enzymatic level by a holoenzyme, glutamate-cysteine ligase (GCL), a heterodimer composed of a catalytic subunit (GCLC), and a modifier subunit (GCLM)
Am J Physiol Endocrinol Metab 299: E62-68, 2010
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