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Notably, L2HG-induced ferroptosis occurs independently of impaired cystine uptake, transsulfuration pathway activity, or increased GPX4-mediated GSH utilization, revealing a non-canonical ferroptosis mechanism driven by enhanced GSH catabolism
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However, the relationship between lactic acid levels and ME/CFS is still being investigated, as another study reported reduced lactic acid levels in urine, along with lower pyruvate and alanine (187)
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How conformational changes influence inflammatory properties
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