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In isolated rat aorta preparations, the peptide produced concentration-dependent vasodilation that was endothelium-dependent and abolished by L-NAME, and co-immunoprecipitation work traced the response to phosphorylation of Src, caveolin-1, and endothelial nitric oxide synthase, with reduced binding between caveolin-1 and eNOS [1]

Some users find they respond well to lower doses and never need to reach the peak phase amounts

Its whether youre getting enough of it and whether the supplement youre taking (if youre taking one) is actually getting into your cells