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JZP-458 was well tolerated in phase I clinical trials

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Moreover, we can conclude from the present study that the positive inotropic effects of dulaglutide in the human heart are mediated via GLP1-R because the the positive inotropic effect of dulaglutide were reversed by exendin(939), an antagonist at GLP1-R (Serre et al

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For carefully selected patients, though, they may be a powerful addition to standard therapy

This does not mean every patient with a medical concern is automatically excluded