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Similar to this process, mono- and polyunsaturated long-chain fatty acids stimulate GLP-1 release via certain free fatty acid receptors
Suppressing glucagon (which raises blood sugar)
The inter-organelle complex formed by endosomal GLP-1R, ER VAPB and SPHKAP triggers a pool of ERMCS-localised cAMP/PKA signalling via the formation of a PKA-RI biomolecular condensate which leads to changes in mitochondrial contact site and cristae organising system (MICOS) complex phosphorylation, mitochondrial remodelling, and -cell functional adaptation, with important consequences for the regulation of -cell insulin secretion and survival to stress
Little work has previously been undertaken to evaluate safety with long-term efficacy and cardio-renal benefits of such therapies
Do not jump straight to a higher dose
Many standard users land at 2-5 mg weekly, 6-9 mg is high-efficacy territory, and 10-12 mg is deliberate escalation
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