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The non-enzymatic mitochondrial protein hyperacetylation induced by the increased availability of mitochondrial acetyl-CoA avoids the risk associated with mitochondria overfeeding, restraining the activity of the respiratory complex I and modulating CACT activity
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10.14348/molcells.2016.2368 287 RicciC.MarzocchiC.BattistiniS
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Immunomodulation and inflammation: Role of GLP-1R and GIPR expressing cells within the gut
Table 1 summarizes some of the most representative used donors for NO and H 2 S
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